There is certainly an Affiliation among greater troponin concentrations and remaining ventricular hypertrophy.195 It is thought which the subendocardium might have impaired perfusion due to elevated wall stress, concomitant endothelial dysfunction and, frequently, enhanced myocardial oxygen desire.

The answer lies in The very fact that there are various isoforms of each troponin subunit which happen to be encoded by distinct genes, several of which can be expressed selectively in cardiac muscle. Although TnI and TnT have unique cardiac and skeletal isoforms, they share a standard isoform of Tn

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